Bryan Johnson· Author
a stereoisomer of 17β-E2 that is at least 200-fold less active as a hormone against ‘classical’ estrogen receptors (ER) ER beta (but not necessarily ER alpha, the other classical receptor)
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
a stereoisomer of 17β-E2 that is at least 200-fold less active as a hormone against ‘classical’ estrogen receptors (ER) ER beta (but not necessarily ER alpha, the other classical receptor)
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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