The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
and then comt is a little different to um tea as you know is the one that is gonna methylate the catecholamines and estrogen to detoxify it so if we have a slow comt and we're not methylating the estrogen to get rid of it then that's a whole separate story
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
and then there are two snips or genes that are major players in this the MTHFR gene and the COMT gene which I'm a homozygote for MTHFR so if you have any of these snips you could be a poor meth later to begin with and so if this pathway doesn't work and that's what would increase your risk of estrogen dominance and estrogen it cancers