The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
Now, this is important because as we start to think about the different ways to modulate the sex steroid hormones, so-called optimize the hormones, keeping levels of IL-6 low is going to be important for them to exert their effects
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
IL-6 doesn't just travel to the gonads and shut down the gonads, it actually has ways to interact with some of the receptors that the steroid hormones, estrogen and testosterone, bind to, and impact those receptors so that the sex steroid hormones can't have their effect.