Paul Saladino· MD
the reverse t3 is sort of standing in front of the T cells and blocking at t3 cells and blocking it from entering
The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
the reverse t3 is sort of standing in front of the T cells and blocking at t3 cells and blocking it from entering
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
and the problem is that reverse t3 blocks cellular binding sites for this hormone
the problem is when it over converts into reverse t3 so i hope that that cleared that up and the way i like to describe it and you know some people be like this isn't science it's a metaphor for people who aren't doctors to understand this which is you can kind of see a reverse t3 problem like a guard standing in front of your cell not allowing any of the t3 you're taking or your thyroids making to kind of get in and do its job