The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
it does it by modifying cortisol so the molecule cholesterol can be converted into testosterone or estrogen but there's a competition whereby the cholesterol will turn into cortisol and not test testosterone or it'll turn into cortisol and not estrogen if stress levels are too high
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
and the other thing is cortisol so if you have hypercortisolism packed testosterone production directly and indirectly what is hypercortisolism cortisol that's all this yeah in medicine we have to come up with fancy ways to say the short that makes so heal smarter so if you have too much cortisol one you have a you have a shunt right you have something called the pregnenolone shunt or steal where you start to steal pre-curved SURS of testosterone to make more cortisol so that's depleting you of substrate to make this but the I think perhaps the more the more sinister issue is these high levels of cortisol impair you from entering what's called delta-wave sleep which is where you make the FSH and LH to make the testosterone you only make this stuff when you're sleeping