The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
Well, the authors make some good arguments as to why it's not the shift in pH per se, or the shift in carbon dioxide levels in the blood, but rather it's the release of epinephrin. And there's some good reason to believe why that's the case.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
How is the breathing leading to these shifts in, or I should say reduction in inflammatory cytokines and an increase in the liberation of these anti-inflammatory cytokines? This is beyond the scope of this discussion, but that it's actually the release of epinephrine, AKA adrenaline, that's causing this reduction in inflammation.
This is, to me, one of the most concrete examples of a zero-cost tool that bridges the activation of the nervous system through breathing with the immune system by way of releasing adrenaline and thereby reducing the terrible effects or feelings of lousiness from a, in this case, an E. coli infection.
And then the breathing, the controlled breathing, increases the epinephrine and the adrenaline, which then cause all the anti-inflammatory response and then it decreases carbon dioxide, which then stops the pain.
Norepinephrine is increased by the cold, and it suppresses the cytokine production, but the epinephrine from the breathing does it even more robust.