The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
when we have mitochondrial dysfunction we are not joining that ATP to the phosphate there's extra phosphate around that phosphate can diffuse into the cellular matrix outside of the mitochondria and activate Pro inflammatory mediators through phosphorylation
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
those phosphates one of the things I can do is join in to another thing in this case an inflammatory Pro mediator or Pro inflammatory mediators and say that activates that pro-inflammatory mediator