Paul Saladino· MD
insulin can directly increase the activity of epithelial sodium channel sodium phosphate transporter and the sodium hydrogen exchange type 3 and the sodium potassium atpase
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
insulin can directly increase the activity of epithelial sodium channel sodium phosphate transporter and the sodium hydrogen exchange type 3 and the sodium potassium atpase
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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so the majority of renal sodium transporters are controlled by insulin in the kidney several groups using primary cell culture have demonstrated that insulin can directly increase activity of the epithelium sodium channel sodium phosphatase co-transporter the sodium hydrogen exchanger type 3 and the sodium potassium atpase et cetera et cetera