Paul Saladino· MD
these postprandial glucose excursions are probably more damaging for us perhaps from this oxidative stress perspective and in terms of cellular damage
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
these postprandial glucose excursions are probably more damaging for us perhaps from this oxidative stress perspective and in terms of cellular damage
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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what we know is that the the median amplitude glucose excursions or the mages are probably worse than the actual the actual overall level of glucose so in terms of glycosylation or glycation of proteins in the human body pre-diabetes diabetic phenotypes these postprandial glucose excursions are very damaging and often missed if we're only checking am fasting glucose
and if people really had an excursion would be how much the glucose will rise postprandial e and anything above 20 or 30 is probably too much input for a postprandial glucose excursion