The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
but I love that you put that in the context of inflammation because we know that inflammation independent of diet can cause insulin resistance and that that the inflammation gives sort of the context for the adipocyte to say what is the adipocyte threshold at which that the dipa site is going to become insulin resistant
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
so if you have anything I think there's Krawczyk causing a chronic systemic inflammatory state then that is going to modify how much fat your cells are willing to take up before they start becoming insulin resistant
so if you have anything i think that's causing causing a chronic systemic inflammatory state then that is going to modify how much fat your cells are willing to take up before they start becoming insulin resistant