We can't find evidence that holds up here. Proponents are reasoning from mechanism or analogy rather than direct human data, and the most credible skeptics raise objections we can't dismiss.
Metrics like triglyceride/HDL ratio, fasting insulin, and c-peptide are CRUCIAL to understanding one's risk for cardiovascular disease. Yet these metrics are broadly ignored by physicians today who equate an "elevated" LDL on a carnivore or ketogenic diet with one from someone with metabolic dyslipidemia.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
unfortunately most in the medical community only look at LDL they won't look at triglyceride to HDL ratio they won't look at inflammatory markers and they definitely won't look at fasting insulin and measures of insulin sensitivity
so many times when we're thinking about lipids and this is again a little bit off the topic of what we were talking about postprandial hypertriglyceridemia but if we're talking about lipids it's all about context and I really get disappointed when within the mainstream people get hyper focus on LDL without thinking about it in the context of HDL triglyceride HDL ratio inflammatory markers insulin sensitivity markers like fasting insulin or you know toast Amin etc etc