The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
when you eat a meal you release two incretin hormones • gp1 and Gip • glucon like peptide one and glucose dependent insulin trophic polypeptide those two incretin hormones when you eat a meal account for about 70% of the insulin that is released in resp response to the meal so now what is the problem is the problem that you don't release enough glp1 in Gip or is it that your beta cell is refractory to the glp1 in Gip well it's the later
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
You're going to put more GLP-1 in the system. you're going to overcome the resistance at the beta cell. You make more insulin. You now have better glucose control.