We can't find evidence that holds up here. Proponents are reasoning from mechanism or analogy rather than direct human data, and the most credible skeptics raise objections we can't dismiss.
if the mutations with fh are affecting the immune cells that are directly causing or creating atherosclerosis then we have an imperfect system there are many variables being modified there fh is a very poor not a good model system for hypercholesterolemia because of these mutations and their many effects on lipid metabolism
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.