The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
does this also account for like insulin independent glucose uptake like for like via exercise and things like that that does not require insulin uh like does does it affect that peripheral insulin resistance affect the glucose uptake when it is independent of insulin i don't think so i'd have to do more research on it
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
our fat cells determine the insulin sensitivity of our muscles the fat cells are the director they're the conductor at the front of the train and when the fat cells release certain lipokines which are like hormones that are lipids into the blood they signal to the muscles to become insulin resistant but that term insulin resistant is confusing because we say insulin resistance is diabetes and then we say insulin resistance happens in keto and there are very different states this one is physiologic insulin resistance not to be confused with pathologic insulin resistance