The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
do we find people who have high LDL and high HDL and low triglycerides which Dave has sort of coined the triad right so we're presupposing that high HDL low triglycerides are an indication of insulin sensitivity and relative metabolic health
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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on top of that on top of that usually just from those lipids alone I can already guess their blood pressure is low their fasting insulin is low
and furthermore if you stratify people into groups of people who have high HDL and low triglycerides there is no evidence that rising LDL is associated with cardiovascular risk
there's some really great epidemiologic evidence and there's some really great interventional evidence to suggest that an elevated LDL with a high or moderately high HDL and low triglycerides is not the same thing physiologically as a high LDL with a low HDL and high triglycerides
you virtually all of your insulin resistant patients for getting ascribe disease to take OB its LDL particle mediated because everybody who's not on a drug or a serious diet who had a triglyceride HDL cholesterol axis abnormality has astronomically poby
if i saw what was up the first thing i'm saying up i'm dealing with an insulin resistant person because that would be the most common cause not some genetic triglyceride problem