The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
So it may not raise your LDL per se, but it will lower your good cholesterol, or HDL, which-- there's good and bad HDL. So it's a little more complicated than that. But it'll raise your triglycerides, and it will raise your Apo B.
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it can be driven by cholesterol synthesis and we can talk about that because it's going to factor into you know dietary choices for example so how certain dietary patterns will lead to higher LDL than others it's impacted by cholesterol reabsorption so the we can talk about what the life cycle of cholesterol is but again it's um you know we make it and we reabsorb it and it gets circulated it can have to do with triglyceride burden so this is where insulin resistance really factors in to how apob can go up and ultimately it comes down to clearance
so the first mechanism in which we see a very clear relationship between diet and apob is the higher the burden of triglycerides the higher the burden of apob