We can't find evidence that holds up here. Proponents are reasoning from mechanism or analogy rather than direct human data, and the most credible skeptics raise objections we can't dismiss.
on previous podcasts i've gone much further down this rabbit hole and talked about glycogen storage disease 1a a condition with elevated levels of ldl with no elevation in rates of atherosclerosis so if april b is causative atherosclerosis how can people with glycogen storage disease 1a not have higher rates of atherosclerosis when they have elevated levels of ldl
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.