The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
So because the new view that the modern understanding is that it's not adrenaline released systemically kind of bathing all your fat tissue, but rather its neurons releasing adrenaline epinephrin locally that in theory, exercise that stimulates the release of epinephrin or exercise coupled with things like shiver or low grade shaking movement, or the NEAT, the non-exercise activity thermogenesis could in theory lead to local enhancement of mobilization of fat tissue.
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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If you become very adapted to a particular pattern of exercise, whether or not you're subcaloric or not, you're on maintenance calories are not, you are oxidizing some fat always, and you're utilizing the neurons that innervate fat in a regular way. And pretty soon this innervation is going to shut off, because there's no reason why this neural innervation of fat should continue to release epinephrin unless you give it a strong stimulus like cold or the fidgeting or in this case to do novel forms of exercise.
Those subtle movements of our core musculature, not just the core but all our limbs and our musculature, those low-level movements, they trigger epinephrine release from these neurons. They stimulate the mobilization of fat and then that fat is oxidized at higher rates.