The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
what if we do an experiment where we connect the circulations of these two different strains of obese mice and test the hypothesis that maybe there's a circulating factor a hormone that is produced by one of these strains and that controls appetite
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
this parabiosis experiment revealed something really important when they lesioned the ventromedial hypothalamus in one of the rats that was connected to the other rat that rat got very very fat it's just really obese the other one however got very thin it actually lost weight so what does this tell us this tells us that there's something in the blood that's being exchanged between the two animals because it was their blood supply that was linked and that tells us that there's hormone or endocrine signals that are involved in the desire to eat and hunger and appetite