Andrew Huberman· PhD
So with optogenetics, we were able to provide in animals without being on any ketamine or any drug and we could cause the dissociative state by playing in a precise pattern of activity.
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
So with optogenetics, we were able to provide in animals without being on any ketamine or any drug and we could cause the dissociative state by playing in a precise pattern of activity.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
But we were able to manifest this in mice, administering these dissociative agents in mice. We could make them still able to detect stimulus, but not care that it was happening.