The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
excessive gluconeogenesis causes hepatic insulin resistance Paradox and it's sequela the sequela which is a fancy word for the downstream effects are non-alcoholic fatty liver disease well what causes excessive gluconeogenesis insulin resistance causes excess gng so it's not fructose causing uh fatty liver disease it's elevated cortisol insulin resistance and then you get fatty liver disease
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.