The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
they also break aldehyde or atp synthase aka complex five so the point of the electron transport chain is to take substrates like glucose or fats and turn them into atp hne breaks the last step of that chain right so it breaks complex five atp synthase uh impairing the production of atp out of the mitochondria
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
it's a mitochondrial regulator along with reactive oxidative species when you and peter were talking about this this is a study i got from peter h a also causes um the down regulation of atp production in the mitochondria and you know for similar reasons it seems to be a signal that there's excess oxidative stress going on in the mitochondria along with uh reactive oxidative species