Peter Attia· MD
what we've observed is at least in preclinical models when you have severe mitochondrial decline breathing thinner air appears to be beneficial
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
what we've observed is at least in preclinical models when you have severe mitochondrial decline breathing thinner air appears to be beneficial
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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perhaps the single most insightful and interesting thing that we talked about that completely blew my mind was the role of hypoxia as a treatment I want to repeat that again the role of hypoxia oxygen DEP evasion as treatment
five years ago in some of these mouse models or cellular models so we had zero ways of alleviating mitochondrial disease in a dish or in a mouse now we have in our laboratory we can use and again in a preclinical way we can use hypoxia and it actually helps to restore cellular function and longevity and health span in mouse models of mido disease