The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
Now, if there's oxygen available, if there's sufficient oxygen there, what can happen is that pyruvate can be brought to the mitochondria and through a whole set of things that you probably don't want to hear about right now like the electron transport chain and citric acid cycle. What happens is it's broken down and you get 28 to 30 ATP, which has a lot of ATP.
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
alternatively if you if oxygen is plentiful and more importantly if the demand for ATP is not excessive you can shuttle that pyruvate into acetyl co a in the mitochondria and you can generate over 30 units of ATP right