The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
the nmda receptor's typical function okay so when there's no ketamine in the body or brain is to detect abnormal levels of neural activity and in doing so recruit changes to cells receptors Etc literally change the neurons in ways that allow them to respond to that activity in the future without having to be under such big metabolic demand and they do that by recruiting more receptors Etc
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
I do believe it's important to to understand the key components of neuroplasticity which is this remarkable feature of our brain and nervous system that we all have right this ability to change our own brain circuits no other organ in the body as far as we know can direct its own changes but we can direct our own brain changes and the nmda receptor is absolutely critical for that