The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
it looks like there's plenty of evidence to suggest that humans don't need to make a whole bunch of butyrate both because in ketosis you can use beta-hydroxybutyrate from the circulation directly and that the other uh short-chain fatty acids can serve as an intermediary fuel or as a an alternate fuel and might even be higher octane because they have the same effects in the gut that triggering all the same receptors
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
basically in that study the people on the animal-based diet still produce butyrate so i think some of that is probably from intraconversion of isobutyrate to butyrate in the gut so there are microbes who can kind of interconvert and there's also evidence that certain microbes can even convert ketones um which are released into the gut lumen into butyrate so they're basically converting beta hydroxybutyrate to butyrate in the gut lumen as well