Rhonda Patrick· PhD
it was quite clear that omega-3 in human studies and in animal studies seems to blunt the lps the postprandial and endotoxemia as it's called or the lps response after a meal
The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
it was quite clear that omega-3 in human studies and in animal studies seems to blunt the lps the postprandial and endotoxemia as it's called or the lps response after a meal
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saturated fat and then and then polyunsaturated fat in the form of omega-3 has been shown to lower lps leakage from the gut
Because omega-3 actually in um decreases LPS release after a meal. So in this study here, there is a low fat and a highfat meal. Typically higher fat fat meals do increase more LPS because fat can kind of lice this the gut cells and be an irritant on the gut. But omega-3 in the green at at every point um at the baseline it seems as though the omega-3 group actually had a higher LPS. But after the meal, you can see it's it's suppressing the release of LPS and endotoxin into your bloodstream.
And it's very powerful um and important here because now we're talking about not only is omega-3 fatty acids reducing inflammation at the level we already discussed with these specialized pro-resolving mediators and the protectins and the resolins. But even at the level of the gut, and we know this, we know that from animal studies looking at mechanism that omega-3 fatty acids in the gut, when you take the omega-3 fatty acids, they're working on site. They're working at the first, you know, organ that sees it, which is your gut. And it's