The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
i think there are a few things more important than nitric oxide at the level of the endothelium and we know that pleiotropic mechanism of statins probably has to do with nitric oxide things that decrease nitric oxide in the endothelium seem to create more atherosclerosis things that increase nitric oxide seem to be beneficial this is a completely ldl independent mechanism
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
what if the actual effect of statins the actual protective benefit of statins in secondary prevention and people who have had a heart attack comes from improving nitric oxide synthesis at the level of the endothelium