We can't find evidence that holds up here. Proponents are reasoning from mechanism or analogy rather than direct human data, and the most credible skeptics raise objections we can't dismiss.
So the increase seems to be proportional to the differential. So if you rapidly increase ketones 2 millmer and if you stay under 2 millmer then you don't get the spike in insulin. But if you consume it and you get above 1.5 to 2 millmer, at least in me and a few other people that did this, then you see this counter regulatory dump in insulin.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
And that would also explain when people drink a large dose of a ketoester, their glucose levels go down. And it's a bit of a scary situation because I know people have gotten themselves into the situation is that when you drink the ketoester about two hours later or thereabouts, you can be hypoglycemic and also hypoctootic, which means your ketones come up, you utilize them as fuel, but you've released insulin peripheral glucose disposal, and then you get into a point where you're running, for example, and then you tank because you don't have and you can it could trigger a headache. it could I mean as it does with me uh if you take a large dose