The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
So when we smoke or when we ingest nicotine in any other way, you activate these POMC neurons, you suppress appetite, but in part you do that by actually limiting the impulse to chew.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
It turns out that when nicotine gets into the bloodstream and then into the brain, some of that nicotine binds to nicotinic alpha four beta two containing nicotinic receptors. Again, these sub units of receptors, but basically the nicotine parks there. And as a consequence, these POMC neurons increase their electrical activity and appetite is suppressed.