The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
what the new thinking might new thinking is the body uh has it in a diabetic situation has a hard time taking up glucose because of uh insulin signaling and the glute 4 mechanism is not working very well so think about lactate not as a stress but as a strain so now we're going to bypass this inhibition of glucose uptake we're going to provide actually the preferred carbohydrate
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
you look at lactate levels in you look at lactate levels uh in individuals at rest who have type 2 diabetes versus lactate levels at rest in worldclass athletes there's a significant difference and the great irony of that is the very low levels of resting lactate in the athlete mean that at rest they're quite capable of oxidizing fatty acids when sympathetic Drive is low and demand is low and yet paradoxically the individual of type 2 diabetes who would most benefit from fatty acid oxidation is presumably now inhibited in doing so because of those elevated levels of lactate