Rhonda Patrick· PhD
It's a compound that stimulates mitophagy (mitochondrial autophagy), helping clear out and replace damaged or dysfunctional mitochondria.
Verbal pushback — no audited source on record.
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
It's a compound that stimulates mitophagy (mitochondrial autophagy), helping clear out and replace damaged or dysfunctional mitochondria.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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Supplementation with urolithin A improved endurance and improved mitochondrial function in older adults compared to placebo.
Humans given a high dose of urolithin A induced markers of mitophagy and mitochondrial biogenesis — the clearance of damaged mitochondria and growth of new mitochondria — in plasma and skeletal muscle.
Supplementation with urolithin A improves muscle strength, exercise performance, and biomarkers of mitochondrial health including mitophagy.
There's even a recovery-oriented signal in trained athletes.