Bryan Johnson· Author
Levels went from 40 to 67.8 to 110 to 134 in three months (same timeframe as HBOT).
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
Levels went from 40 to 67.8 to 110 to 134 in three months (same timeframe as HBOT).
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The liver converts vitamin D into 25-OH-D (the pre-active form we measure). This is an oxygen-reliant process. More oxygen = faster, more efficient conversion. Increased oxygenation also inhibits CYP24A1, the enzyme that degrades vitamin D.
HBOT may radically improve the bioavailability and systemic efficiency of vitamin D, a master regulator of longevity and cellular function.
In the kidneys, better perfusion supports the conversion of 25-OH-D into the active form 1,25(OH)₂D. This accelerates the activation-utilization cycle.
My Vit D levels spiked 235% post HBOT.