The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
Whether or not somebody has a predisposition to alcoholism or not, whether or not they're experienced drinker or not, when people initially start drinking, there are increases in dopamine, or what we call dopaminergic transmission. Dopamine is involved in motivation, in craving, it creates a sense of well-being, it increases energy, again, typically only at the beginning of alcohol exposure.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
Very soon after, and actually triggered by that increase, is a long and slow reduction in dopamine and serotonin and related molecules in circuits. So basically what you're getting is a blip of feel good followed by a long, slow arc of feeling not so great, which is why, typically, people will drink again and again across the night.