Paul Saladino· MD
what they found essentially was that if you had apoe4 you had a lower risk of developing dementia isn't that interesting especially when it's alzheimer's
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
what they found essentially was that if you had apoe4 you had a lower risk of developing dementia isn't that interesting especially when it's alzheimer's
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what they found was that those who had the apoe4 genotype were protected they had the lowest rates of alzheimer's disease
they found that apoe4 was protective they say here increasing levels of cholesterol and ldl were associated with
they found that apoe4 was protective they say here increasing levels of cholesterol and ldl were associated with an increased risk of alzheimer's disease disease in individuals without the apoe4 allele but not in those with apo e4
what they found was that in these people when they had the apoe4 genotype there was no increase in Alzheimer's disease with increased levels of cholesterol and LDL
what they found was that in these people when they had the apoe4 genotype there was no increase in Alzheimer's disease with increased levels of cholesterol and LDL
people who live in nigeria for example the risk of having alzheimer's disease is substantially less than people who live in the u.s or in japan