The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
the system is supersaturated because there's such a vast quantity of them you're essentially kind of supersaturated and more going in or out when they can freely go in and out this has been said by day spraying it's just not going to be the lever but you also have to say well you have populations where you know they have higher numbers and you know they have the lowest heart disease in the world
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
I believe that at one time some of the 18th the the brownie motion of the LDL particles in and out of the endothelium is probably saturated I don't think there's any more LDL particles and you could do a tracer study to prove this I don't think there are any more LDL particles getting into the seven athelia space at one at three times into the 18th relative to one times ten to the eighteenth
Nobody knows exactly where those LDL particles are going to. You know, if you're making more LDL particles because you're delivering their payload to your muscles and none of them are taking kind of a side detour into your artery wall, that's like a no harm, no foul type situation.