The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
Most of the time, it is things like disruption of calcium that's going to lead to excessive swelling, excessive pressure, and that's going to be then translated as extreme pain. So that's probably explaining more muscle soreness than actually microtrauma.
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all of a sudden we start getting fluid accumulation and now there are what are called nose receptors and you're probably very obviously you're very familiar with these and these are pain receptors what's actually interesting is we don't necessarily know a lot of information about how many pain receptors are in muscle they're not really in the belly in fact this is why I can perform my muscle biopsies and they don't really hurt you mean in the belly of the muscle correct yeah we do have pressure sensors though and so if you change the volume of the tissue you will respond to that very very quickly so by enhancing swelling in the actual muscle that is immediately putting pressure on those pressure receptors if you will that's the signal
Um at the end of the day most of the time exercise induced muscle soreness is a cellular pressure issue and so the more that you can get that out there you have to get the inflammatory signal out of there because that's going to keep putting you back into tissue swelling. So, get the the the the fluid out and then stop the signal that says stop putting more tissue in or fluid in there.