Andrew Huberman· PhD
And there are these bacteria, like Akkermansia muciniphila, mucus-loving. One of its main things it does is actually eat mucus in the gut, that's its lifestyle.
The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
And there are these bacteria, like Akkermansia muciniphila, mucus-loving. One of its main things it does is actually eat mucus in the gut, that's its lifestyle.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
it really is like this Keystone strain for a reason
but the other thing that not not only does acran stimulate uh these L cells to produce gop1 but acran has another role which maybe is very much tied to this — you know which is that uh it it helps regulate the mucin layer and so that mucin layer turns out to be super important for uh if it gets too thin or if it's too thick it's it's it's a real this so-called leaky gut or GI issues that that become a result of not having enough acromania is really that you have too thin of a mucin layer and so you start to get — you know the ability for pathogens to infiltrate and then also obviously for these molecules to secrete out