Andrew Huberman· PhD
And the reason why you have to do that as opposed to somebody who does not have hypothyroidism, is if you have hypothyroidism, then likely your thyroid will not respond to either TSH or hCG.
The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
And the reason why you have to do that as opposed to somebody who does not have hypothyroidism, is if you have hypothyroidism, then likely your thyroid will not respond to either TSH or hCG.
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
For example, we do this in during pregnancy. Uh, a a woman with hyperyroidis that becomes pregnant, we want to treat the woman with antiyroid medication, but we want to give as little as possible. So because the drugs cross the placenta and they can cause hypothyroidism in the fetus. So you'll let the TSH be suppressed. I want to give the amount of drug that's going to keep the free T4 in the upper limit of normal. So my reference becomes the free T4 and not the TSH anymore.