The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
atheroma related atherosclerosis does not occur independent of insulin resistance which means if you are insulin sensitive you're fine if your ldl goes from 120 to 180 but you're fasting insulin which is the best measure of insulin sensitivity remains low less than 5 micro iu per ml or i would prefer it even less than three micro io per ml
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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atheroma related atherosclerosis does not occur independent of insulin resistance which means if you are insulin sensitive you're fine if your ldl goes from 120 to 180 but you're fasting insulin which is the best measure of insulin sensitivity remains low less than 5 micro iu per ml or i would prefer it even less than 3 micro io per ml
is it possible that LDL is involved in the atherosclerotic process when arteries are damaged yes does that mean LDL initiates it no and I think that we can mitigate and minimize damaged arteries by being insulin sensitive which allows healing turnover of the endothelium in a proper way
we know that if you're insulin sensitive your risk of these things is essentially so much infinitesimal it's so much smaller when if you're but if you're diabetic or you're metabolically unwell your risk is massively increased because your wound healing is bad