The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
FOXO3 in turn upregulates pathways related to DNA repair and clearing out of these senescent or dead cells, which is known to be important for various aspects of maintaining cognition and other aspects of maintaining health.
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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FOX3 in turn upregulates pathways related to DNA repair and clearing out of these senocent or dead cells which is known to be important for various aspects of maintaining cognition and other aspects of maintaining health.
foxo3 increases the expression of genes that are involved in DNA repair so that the damage is repaired and never forms a mutation foxo3 also increases the expression of genes that kill damaged cells so they can't form cancer
foxo 3 activates genes that are involved in DNA repair that repair that damage before it ever can form a mutation
well foxo3 activates genes antioxidant genes that prevent that damage from ever hitting the cell they also foxo3 activates genes that if the cell does become senescent and is damaged genes that are involved in um autophagy and clearing away that damage