Andrew Huberman· PhD
and by also disrupting the packaging of dopamine and norepinephrine into vesicles themselves what ends up happening is that there's a buildup of a lot more dopamine and norepinephrine in the presynaptic terminal
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The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
and by also disrupting the packaging of dopamine and norepinephrine into vesicles themselves what ends up happening is that there's a buildup of a lot more dopamine and norepinephrine in the presynaptic terminal
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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what Adderall does and what other sympathomimetics do is to inhibit or disrupt the action of those Transporters and the net consequence of that is that when dopamine and or epinephrine are released into the synapse more of it is allowed to stick around and to binder receptors on the postsynaptic cell than would be the case if Adderall or the other stimulant were not present in the system