The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
evolutionarily inappropriate consumption of grains by these monogastric animals chicken pork turkey etc duck leading to elevated levels of linoleic acid in the fat elevated levels of linoleic acid in the muscle tissue elevated levels of linoleic acid in the eggs and that parallels very closely with the mycotoxins
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if you're eating chicken for the majority of your diet that chicken is fed corn and soy and if you can find corn and soy free chicken they're probably still fed grains all those grains are going to be moldy and they're going to accumulate mold toxins and i'll just add this little story
these animals are fed grains they're going to be higher in aflatoxin in other xerolino and other mycotoxins because this grain is accumulating uh mold and that is going to bioaccumulate in these animals
chickens are monogastric like humans and pigs when we eat more lenic acid we store more lenic acid when a chicken is fed corn and soy there's more lenic acid in the eggs in the yolk and in the chicken fat than there would be in a wild chicken