Paul Saladino· MD
polyunsaturated fatty acids may also have a mechanism of interfering with triodo thyronine that is t3 at the level of the thyroid receptor
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
polyunsaturated fatty acids may also have a mechanism of interfering with triodo thyronine that is t3 at the level of the thyroid receptor
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The results indicate that the unsaturated long-chain fatty acids, linoleic acid, are potent inhibitors of thyroxine binding to thyroxine-binding globulin, whereas saturated fatty acids have little or no effect on thyroid binding.
there's evidence that linolic acid and oleic acid both negatively affect The Binding of T3 to its nuclear receptor they both negatively affect the conversion of T4 to T3 so the conversion of thyroxin to triota thyronine T3 being triota thyronine that's the active form of thyroid hormone and they both negatively affect The Binding of thyroxin and T3 to thyroid binding globulin
there's evidence that linolic acid and oleic acid both negatively affect The Binding of T3 to its nuclear receptor they both negatively affect the conversion of T4 to T3 so the conversion of thyroxin to triota thyronine T3 being triota thyronine that's the active form of thyroid hormone and they both negatively affect The Binding of thyroxin and T3 to thyroid binding globulin
They can interfere with the production of thyroid hormones. They can interfere with the release of thyroid hormones. They can displace thyroid hormones and vitamin A from trans thyroid, which is one of the carrier proteins for thyroid hormones. And they can potentially interfere with T3 binding to its receptor within the cells or at a cellular level.