Andrew Huberman· PhD
Ingesting fructose shifts our hormone system, and as a consequence, our neural pathways within our brain, the hypothalamus, to be hungrier regardless of how many calories we've eaten.
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
Ingesting fructose shifts our hormone system, and as a consequence, our neural pathways within our brain, the hypothalamus, to be hungrier regardless of how many calories we've eaten.
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Fructose reduces the activity of the hormones that reduce Ghrelin . And so the net consequence of that is that fructose increases Ghrelin.
fructose and specifically fructose, has the ability to reduce certain hormones and peptides in our body whose main job is to suppress Ghrelin.
Ingesting fructose shifts our hormone system and as a consequence our neural pathways within our brain the hypothalamus to be hungrier regardless of how many calories we've eaten.
One of the key distinctions between glucose and fructose is that fructose most likely cannot directly access the brain. It actually needs to be converted into glucose in the liver. And the way that conversion occurs feeds back to a set of hormones and neural pathways that we talked about earlier which have a lot to do with appetite. And to just summarize what is now a lot of very solid data, fructose and specifically fructose has the ability to reduce certain hormones and peptides in our body whose main job is to suppress ghrein.