The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
The increases in dopamine associated with sweet taste and/or blood glucose elevating foods and drinks, cause activation of the nucleus accumbens. That's not surprising. Also in the circuit is activation of the so-called arcuate nuclei within the hypothalamus.
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Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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But hypothalamus is sort of the accelerator and the break on eating. And then the nucleus accumbens and dopamine release can be thought of as kind of a nitro boost, if you will, like the kids say. Do the kids say that anymore? Anyway, a nitro boost to increase what we call the gain or the volume of how much you want more of something, okay?
the quotes discuss is glucose stimulates nucleus acumin uh dopaminergic Pathways as well triggering the honic the hedonic feeding and binging fat stimulate dop energic Pathways in the nucleus acumin and also triggers hyperphagia un binging
when we eat sugar the taste buds are activated and it stimulates this dopamine response in the brain that tells us that we like this sugar