The headline is broadly defensible, but the qualifications matter. Effect sizes vary by population, the strongest claims rest on shorter trials, and credible voices push back on how it's typically framed.
the arteries are a higher pressure system and at the branch points there is turbulence which can create some endothelial dysfunction some shredding some shearing stress and that creates the beginning of an atherosclerotic plaque
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Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
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this is why we don't get atherosclerosis in veins but we only see it in arteries in a normal human individual because the arteries are a higher pressure system and at the branch points there is turbulence which can create some endothelial dysfunction some shredding some shearing stress and that creates the beginning of an atherosclerotic plaque
there do not see atherosclerosis plaque formation and veins you only see it in arteries in the native circulation and this is an unhealthy person like a diabetic person is much more predisposed to get atherosclerosis plaque formation cardiovascular disease these are all synonyms they get it in the arteries they don't get it in the veins why is that because the pressure in the veins is so much lower we don't get the turbulence at the bifurcations in the veins so the endothelium is not being damaged