Rhonda Patrick· PhD
For example, a 2021 study found that a 1-standard deviation increase in genetically proxied LDL-c was associated with a 1.2 year lower lifespan and a 28% reduction in the odds of longevity to the 90th percentile age.
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
For example, a 2021 study found that a 1-standard deviation increase in genetically proxied LDL-c was associated with a 1.2 year lower lifespan and a 28% reduction in the odds of longevity to the 90th percentile age.
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Mendelian randomization studies have found that higher levels of low-density lipoprotein cholesterol (LDL-c) are associated with a reduction in lifespan and longevity.
For example, a 2021 study found that a 1-standard deviation increase in genetically proxied LDL-c was associated with a 1.2 year lower lifespan and a 28% reduction in the odds of longevity to the 90th percentile age.
Mendelian randomization studies have found that higher levels of low-density lipoprotein cholesterol (LDL-c) are associated with a reduction in lifespan and longevity.
For example, a 2021 study found that a 1-standard deviation increase in genetically proxied LDL-c was associated with a 1.2 year lower lifespan and a 28% reduction in the odds of longevity to the 90th percentile age.
Mendelian randomization studies have found that higher levels of low-density lipoprotein cholesterol (LDL-c) are associated with a reduction in lifespan and longevity.