Andrew Huberman· PhD
Post-cardio reductions in hunger may be caused by “lac-phe” (comprised of lactate and phenylalanine).
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The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
Post-cardio reductions in hunger may be caused by “lac-phe” (comprised of lactate and phenylalanine).
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lactate is a very powerful appetite suppressant
lactate has powerful effects not just on our body body but on our brain and it is able to impact the activity of neurons in our so-called hypothalamus little marbleized region above the roof of our mouth that contains some of the neurons that control our appetite and our degree of satiety
High intensity exercise potently increases blood lactate levels, which are known to suppress the hunger hormone. In the study, high intensity exercise led to a more substantial increase in blood lactate levels than moderate exercise.
if you do raise lactate it will cross the bloodb brain barrier it will inhibit grin secretion and it will suppress the appetite
it inhibits appetite lactate suppresses gin it works directly in the CNS so an advantage of doing an exercise not like that one you did Peter but getting lactate up to maybe three or four Millar would actually help satiate people
vigorous exercise is anorectic you don't want to eat after you finish a math on or or at least I don't no and and by the way just to project I interviewed George Brooks from Berkeley recently and he shared with me on the podcast something I didn't know before which was that lactates specifically is probably partially driving the anorexia we experience following intense exercise