Andrew Huberman· PhD
Assuming that it's true male pattern baldness, it's related to the gene transcription of the androgen receptor.
The evidence is convergent. Multiple independent sources reach the same conclusion, the underlying mechanism is well-characterized, and even the field's most cautious voices treat it as worth doing.
Assuming that it's true male pattern baldness, it's related to the gene transcription of the androgen receptor.
Every Sunday: the week’s new conflicts and verdict changes — and nothing else.
Native comments, Twitter mentions, and Reddit threads about this claim — surfaced together so the conversation isn't fragmented across platforms.
Bookmarking — the dossier-vs-overview split is the right call. Most of the time I want overview; sometimes I want receipts.
Would love a "what would change this verdict" RSS feed. Sign me up if it exists.
But anyway if you have more repeats, then that gene activates in the cytoplasm and moves to the nucleus and causes gene transcription more often and hair loss more often.
So if you have an extremely sensitive gene, which usually means you have very few CAG repeats, which is basically just a certain, CAG encodes for a certain amino acid, and if you have very few of the repeats, then your androgen receptor gene works better.
but also there's the the um genetic differences in the receptor itself, which is the KAG repeat, which predict >> the binding, >> the the efficiency of its ability to transcribe um the androgen responsive proteins